Droga esteroides y sus efectos

The primary focus on serotonin deficiency as the main cause of depression has created treatment failure in many depressed and addicted patients. Other happy (excitatory) neurotransmitters are often ignored and some patients become more depressed when treated with medication. The classic depressive disorder patient who presents to Florida Detox ® is Susan, a 42-year-old professional female who seeks medical attention for depression from her local physician. Dr. Jones immediately assumes that she would benefit from a serotonin enhancer such as Paxil, Prozac, or Lexapro.  If indeed this patient suffers from low serotonin levels, her depression should respond within 2-4 weeks of treatment with the serotonin enhancer. Typically, prescribed a medication like Paxil (20 mg per day), she returns one month later insisting her depression is worse. Dr. Jones raises the Paxil to 40 mg per day. Frequently these patients are prescribed extremely high doses (60 mg to 80 mg per day) in the physician’s effort to conquer the problem. Unfortunately Dr. Jones disregards the continuous report from the patient that they are not feeling any better and may actually feel more depressed. What is the problem? If serotonin is unilaterally elevated above normal levels with the mediation, the brain will down regulate production of dopamine. This makes the patient with dopamine deficiency even more dopamine deficient. These patients will typically begin to self medicate with dopaminergic drugs like Percocet, Vicodin, or OxyContin to counteract the decreased production. All of these drugs produce increased dopamine activity in the brain’s pleasure center (nucleus accumbens). When these patients are accurately diagnosed with their genetic dopamine/glutamate deficiency and treated with appropriate dopamine/glutamate enhancing medication, they quickly experience cessation of their depression and lose the craving (psychological and biochemical) for drugs and alcohol. Treatment results in better relapse statistics with the application of this scientific approach to addiction and depression. Unilateral elevation of serotonin without dopamine level protection will result in markedly elevated prolactin levels. Prolactin will increase appetite and decrease sex drive.  When dopamine levels are enhanced to normal levels, sex drive will return as will better appetite control.

La investigación preliminar sugiere que el uso de DHA por parte de las mujeres embarazadas o lactantes podría reducir el índice de masa corporal de los bebés; no obstante, no existen efectos sobre la longitud o el tamaño de la cabeza de los bebés. Se ha descubierto que el consumo de aceite de pescado por parte de la madre durante el embarazo no tiene efecto sobre el desarrollo del idioma en el bebé. Se ha estudiado el efecto de la leche maternizada enriquecida con DHA sobre la seguridad, el crecimiento y el desarrollo de los bebés prematuros y a término. Se necesita más investigación en esta área.

El crecimiento se puede retardar en las personas jóvenes quienes fueron diagnosticadas con EII antes de la pubertad. El consumo pobre de alimentos puede también contribuir a un crecimiento pobre. Por lo tanto, los buenos hábitos nutrimentales y consumo calórico adecuado son muy importantes. El control de la enfermedad con medicamentos o, menos frecuente, la remoción quirúrgica de un área del intestino particularmente mórbida, tiene mayor éxito cuando se mantiene un consumo dietético adecuado.

Droga esteroides y sus efectos

droga esteroides y sus efectos

Media:

droga esteroides y sus efectosdroga esteroides y sus efectosdroga esteroides y sus efectosdroga esteroides y sus efectosdroga esteroides y sus efectos

http://buy-steroids.org