First steroid cycle chart

Hugh Jackman Steroid Cycles There was a lot of talk after the last X-men movie about the amazing shape Hugh Jackman was in.  Many questions arose, such as: was Hugh Jackman taking steroids?  Was a huge Hollywood star using anabolic steroids to look better for a movie?  The likely answer is, YES!  Of course Hugh Jackman was using anabolic steroids, and if you look below you can clearly see his pictures reflect some steroid use.  




What was Hugh Jackman 's steroid cycle?
Likely, he used a combination of fast acting anabolic steroids during his shorter cycles.  In fact, we believe he only used  4 AAS to prepare for the movie X-men.  Trenbolone, Anavar, Clenbuterol and Winstrol.  A combination of steroids, a cycle, which would be effective for almost anyone, but especially someone who needs to shed fat and put on lean, hard muscle mass.  


Week Trenbolone Anavar Clenbuterol Winstrol 1 75mgs/day 40mgs/day 40mcg /day 50mgs/day 2 75mgs/day 40mgs/day 40mcg /day 50mgs/day 3 75mgs/day 40mgs/day 40mcg /day 50mgs/day 4 75mgs/day 40mgs/day 40mcg /day 50mgs/day 5 75mgs/day 40mgs/day 40mcg /day 50mgs/day 6 75mgs/day 40mgs/day 40mcg /day 50mgs/day 7 75mgs/day 40mgs/day 40mcg /day 50mgs/day 8 75mgs/day 40mgs/day 40mcg /day 50mgs/day
DISCLAIMER: This article is for entertainment purposes only! We do NOT believe Hugh Jackman used any performance enhancing drugs, rather this is an easy formula for one to look like Hugh Jackman.


Clenbuterol for men is a very popular product, used by bodybuilders, athletes and men that wish to slim down and to shape their muscles. The so called Clen for men is a substance that is often mistaken for steroid, though in fact it is not a steroid. Clenbuterol for men is a drug that falls in the group of the beta-2-agonists, which has certain similarities with the stimulant drugs. These are not weight loss steroids for men, yet the Clenbuterol pills can increase the heart rate, the perspiration and the blood pressure just like the amphetamines and the adrenalin can do. Read More»

Prostaglandins and proteolytic enzymes, such as collagenase and plasmin, are increased in response to LH and progesterone. Although the precise mechanism is not known, proteolytic enzymes and prostaglandins are activated and digest collagen in the follicular wall, leading to an explosive release of the oocyte-cumulus complex [31]. Prostaglandins may also stimulate ovum release by stimulation of smooth muscle within the ovary. The point of the dominant follicle closest to the ovarian surface where this digestion occurs is called the stigma. There is no evidence to support the theory that follicular rupture occurs as a result of increased follicular pressure, although precise measurements precisely at rupture have not been performed [32]. In a recent report, laparoscopic visualization of human ovulation during an operative procedure was documented. The authors report visualizing a follicular area called the stigma which was protruding like a bleb from the surface, containing viscous yellow fluid evaginating into the peritoneal cavity [33]. In humans, ovulation probably occurs randomly from either ovary during any given cycle. Of interest, some studies have suggested that ovulation occurs more commonly from the right ovary and right sided ovulation carries a higher potential for pregnancy [34]. The concentrations of prostaglandins E and F series and hydroxyeicosatetraenoic acid (HETE) reach a peak level in follicular fluid just prior to ovulation [35, 36]. Prostaglandins may stimulate proteolytic enzymes while HETEs may stimulate angiogenesis and hyperemia [37]. Patients treated with high dose prostaglandin synthetase inhibitors such as Indocin, can have a block in prostaglandin production and effectively block follicular rupture [38-40]. This gives rise to what is known as the luteinized, unruptured follicle syndrome and it presents in fertile and infertile patients equally [41]. Therefore, infertility patients are advised to avoid taking prostaglandin synthetase inhibitors, as well as cyclo-oxygenase (COX) inhibitors, especially around the time of ovulation [40]. A schematic diagram illustrating the proposed mechanisms involved in follicular rupture is presented in figure 7 .

Steroid isolation , depending on context, is the isolation of chemical matter required for chemical structure elucidation, derivitzation or degradation chemistry, biological testing, and other research needs (generally milligrams to grams, but often more [37] or the isolation of "analytical quantities" of the substance of interest (where the focus is on identifying and quantifying the substance (for example, in biological tissue or fluid). The amount isolated depends on the analytical method, but is generally less than one microgram. [38] [ page needed ] The methods of isolation to achieve the two scales of product are distinct, but include extraction , precipitation, adsorption , chromatography , and crystallization . In both cases, the isolated substance is purified to chemical homogeneity; combined separation and analytical methods, such as LC-MS , are chosen to be "orthogonal"—achieving their separations based on distinct modes of interaction between substance and isolating matrix—to detect a single species in the pure sample. Structure determination refers to the methods to determine the chemical structure of an isolated pure steroid, using an evolving array of chemical and physical methods which have included NMR and small-molecule crystallography . [2] :10–19 Methods of analysis overlap both of the above areas, emphasizing analytical methods to determining if a steroid is present in a mixture and determining its quantity. [38]

First steroid cycle chart

first steroid cycle chart

Steroid isolation , depending on context, is the isolation of chemical matter required for chemical structure elucidation, derivitzation or degradation chemistry, biological testing, and other research needs (generally milligrams to grams, but often more [37] or the isolation of "analytical quantities" of the substance of interest (where the focus is on identifying and quantifying the substance (for example, in biological tissue or fluid). The amount isolated depends on the analytical method, but is generally less than one microgram. [38] [ page needed ] The methods of isolation to achieve the two scales of product are distinct, but include extraction , precipitation, adsorption , chromatography , and crystallization . In both cases, the isolated substance is purified to chemical homogeneity; combined separation and analytical methods, such as LC-MS , are chosen to be "orthogonal"—achieving their separations based on distinct modes of interaction between substance and isolating matrix—to detect a single species in the pure sample. Structure determination refers to the methods to determine the chemical structure of an isolated pure steroid, using an evolving array of chemical and physical methods which have included NMR and small-molecule crystallography . [2] :10–19 Methods of analysis overlap both of the above areas, emphasizing analytical methods to determining if a steroid is present in a mixture and determining its quantity. [38]

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