Steroid conversion table pdf

The bulk of published literature indicates that the expression of 5 α -R1 increases and 5 α -R2 decreases in CaP compared to benign prostate and BPH. Umera et al. confirmed for the first time increased expression of 5 α -R3 at the mRNA level in CR-CaP. Godoy et al., confirmed this at the protein level. A validated monoclonal antibody showed that expression of 5 α -R3 was increased similarly in AS-CaP and CR-CaP compared to AS-BP. 5 α -R3 expression was increased in lung, breast, papillary thyroid, and testicular (seminoma and yolk sac) cancers compared to their benign counterparts.

Depending on the number and character of their functional groups, steroid molecules may show diverse reactivities. Moreover, the reactivity of a functional group varies according to its location within the molecule (for example, esters are formed readily by 3-OH groups but only with difficulty by the 11β-OH group). An important property of steroids is polarity —., their solubility in oxygen-containing solvents (., water and alcohols ) rather than hydrocarbon solvents (., hexane and benzene ). Hydroxyl, ketonic, or ionizable (capable of dissociating to form electrically charged particles) groups in a steroid molecule increase its polarity to an extent that is strongly influenced by the spatial arrangement of the atoms within the molecule.

The mineralocorticoid pathway starts with 21-hydroxylation of progesterone to form deoxycorticosterone (DOC). The enzyme in this reaction, 21-hydroxylase, is encoded by the CYP21 gene. 11 , 12 Deoxycorticosterone is then converted to corticosterone through the action of 11β-hydroxylase. There are two distinct 11β-hydroxylase isoenzymes, both of which are encoded by two genes, CYP11B1 and CYP11B2 . 13 Corticosterone is hydroxylated at carbon 18 to form 18-hydroxycorticosterone, which is transformed to aldosterone by removal of two hydrogens (oxidation) at carbon 18. These two reactions are catalyzed by 18-hydroxylase and 18-hydroxysteroid dehydrogenase, respectively, which are encoded by the same gene, CYP11B2 . Transcription of CYP11B1 is regulated primarily by ACTH, whereas angiotensin II regulates CYP11B2 transcription. 14 , 15 Similarly, the glucocorticoid pathway begins with 17α-hydroxyprogesterone, which is converted to deoxycortisol and subsequently to cortisol by 21-hydroxylase and 11β-hydroxylase, respectively, in the same manner as the conversion of progesterone to corticosterone. A deficiency of 21-hydroxylase, 11β-hydroxylase, or 3β-HSD in the adrenals may result in congenital adrenal hyperplasia and female pseudohermaphroditism, manifested as a masculinized female fetus.

Hello Will,
I have for the first time in the past 4 years been able to reduce my hair fall down to 20-100 strands a day by following a regimen you have described above. I notice small hairs sprouting on the top of my head, that are close to 2 inches now. Although, I have lost a lot of hair in the back of my head that are not as responsive as the top of my head. I am still not sure if i have chronic telogen effluvium or androgenic alopecia because both my parents are close to 60 and have head full of hair. Anyhow, I increased the intake of vegetables/fruits (both raw & juice), flax seeds and coconut oil in the diet and have been having ginger/lemon juice before every meal to increase stomach acid as well. I have also included a probiotic supplement with my second meal and have been mindful about processed sugar, wheat and dairy mostly. If i actively maintain this regimen, do I have a good chance of regaining the thickness? And what is the timeline I can expect to reactivate the dormant hair follicles, 6 months, 1 year or 2 years? By stopping the abnormal hair loss and combining with scalp exercises and rosemary oil massage, can we switch the body to recover the dormant hair follicles or the best i can hope is to maintain what i have left?
I really appreciate all your help.

Steroid conversion table pdf

steroid conversion table pdf

Hello Will,
I have for the first time in the past 4 years been able to reduce my hair fall down to 20-100 strands a day by following a regimen you have described above. I notice small hairs sprouting on the top of my head, that are close to 2 inches now. Although, I have lost a lot of hair in the back of my head that are not as responsive as the top of my head. I am still not sure if i have chronic telogen effluvium or androgenic alopecia because both my parents are close to 60 and have head full of hair. Anyhow, I increased the intake of vegetables/fruits (both raw & juice), flax seeds and coconut oil in the diet and have been having ginger/lemon juice before every meal to increase stomach acid as well. I have also included a probiotic supplement with my second meal and have been mindful about processed sugar, wheat and dairy mostly. If i actively maintain this regimen, do I have a good chance of regaining the thickness? And what is the timeline I can expect to reactivate the dormant hair follicles, 6 months, 1 year or 2 years? By stopping the abnormal hair loss and combining with scalp exercises and rosemary oil massage, can we switch the body to recover the dormant hair follicles or the best i can hope is to maintain what i have left?
I really appreciate all your help.

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